Vitamin regimen for anal cancer

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Helen A. Norman, Ritva R. Wargovich, Elizabeth K. Weisburger, Steven H. It encompasses the AICR position on current issues in nutrition for cancer survivors during treatment and is intended to provide advice about dietary supplements for cancer survivors who are still being treated.
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Ascorbic Acid in Colon Cancer: From the Basic to the Clinical Applications

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Ascorbic Acid in Colon Cancer: From the Basic to the Clinical Applications

Before the twentieth century, colorectal cancer was relatively uncommon however the incidence has risen dramatically especially in the last fifty years. Several risk factors have been proposed including the adoption of westernized diet, obesity and physical inactivity 1 , 2. The majority of colorectal cancer continues to occur in industrialized countries. Therefore, diet optimization could potentially help reduce the incidence of this type of malignancy 4 , 5. Here we review the key evidence for the role of different dietary components and their effect on colorectal cancer prevention and progression. The search was performed for the period As expected, the search yielded an overwhelming abundance of evidence on the association between diet and colorectal cancer.
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Anti-Cancer Supplements

When it comes to dietary supplements, there are far too many options to choose from. Dietary supplements are any kind of vitamins , minerals, herbs , botanicals, and amino acids that you can eat or drink. Supplements come in all shapes and sizes, such as:. People take supplements for different reasons. The main function of dietary supplements is to do exactly what the name implies — supplement an existing diet.
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Trials of nutritional supplements for cancer prevention must overcome a variety of challenges not shared in the usual paradigm of pharmaceutical agents for prevention of cardiovascular disease. Unlike for cardiovascular disease, for cancer we typically do not have well-established causal risk factors as targets for intervention. Also, for most likely cancer interventions, the expected time to achieve an effect is much longer, more variable, and far less well understood than for cardiovascular disease, and the progression of pathophysiology is much harder—or impossible—to follow, in contrast with imaging for progression of atherosclerosis in cardiovascular disease. Also, cancers at various sites have a wide range of etiologies. The optimal age for intervention, best dose, and duration needed to test nutritional agents for cancer prevention are largely unknown, making null findings hard to interpret.
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